Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3

Bettina Seidl; Behnam Kalali; Markus Gerhard; Johannes Ring; Markus Ollert; Martin Mempel

doi:10.1016/j.jaci.2009.07.028

Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3

Bettina Seidl^*, Behnam Kalali, Markus Gerhard, Johannes Ring, Markus Ollert, Martin Mempel

^*Corresponding author for this work

Research output: Contribution to journal › Letter › peer-review

6 Citations (Scopus)

Original language	English
Pages (from-to)	862-864
Number of pages	3
Journal	Journal of Allergy and Clinical Immunology
Volume	124
Issue number	4
DOIs	https://doi.org/10.1016/j.jaci.2009.07.028
Publication status	Published - Oct 2009
Externally published	Yes

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10.1016/j.jaci.2009.07.028

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Seidl, B., Kalali, B., Gerhard, M., Ring, J., Ollert, M., & Mempel, M. (2009). Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3. Journal of Allergy and Clinical Immunology, 124(4), 862-864. https://doi.org/10.1016/j.jaci.2009.07.028

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title = "Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3",

author = "Bettina Seidl and Behnam Kalali and Markus Gerhard and Johannes Ring and Markus Ollert and Martin Mempel",

note = "Funding Information: Supported by grants from the DFG (Me 1798/2-1 to M. M.), the BMBF (01GC0104 (subproject 4 to M. M. and M. O.), the Wilhelm-Vaillant Foundation (M. M.), and the KKF University Hospital “Rechts der Isar,” Technische Universit{\"a}t M{\"u}nchen. ",

year = "2009",

month = oct,

doi = "10.1016/j.jaci.2009.07.028",

language = "English",

volume = "124",

pages = "862--864",

journal = "Journal of Allergy and Clinical Immunology",

issn = "0091-6749",

publisher = "Elsevier Inc.",

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}

Seidl, B, Kalali, B, Gerhard, M, Ring, J, Ollert, M & Mempel, M 2009, 'Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3', Journal of Allergy and Clinical Immunology, vol. 124, no. 4, pp. 862-864. https://doi.org/10.1016/j.jaci.2009.07.028

Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3. / Seidl, Bettina; Kalali, Behnam; Gerhard, Markus et al.
In: Journal of Allergy and Clinical Immunology, Vol. 124, No. 4, 10.2009, p. 862-864.

Research output: Contribution to journal › Letter › peer-review

TY - JOUR

T1 - Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3

AU - Seidl, Bettina

AU - Kalali, Behnam

AU - Gerhard, Markus

AU - Ring, Johannes

AU - Ollert, Markus

AU - Mempel, Martin

N1 - Funding Information: Supported by grants from the DFG (Me 1798/2-1 to M. M.), the BMBF (01GC0104 (subproject 4 to M. M. and M. O.), the Wilhelm-Vaillant Foundation (M. M.), and the KKF University Hospital “Rechts der Isar,” Technische Universität München.

PY - 2009/10

Y1 - 2009/10

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U2 - 10.1016/j.jaci.2009.07.028

DO - 10.1016/j.jaci.2009.07.028

M3 - Letter

C2 - 19767083

AN - SCOPUS:70449707286

SN - 0091-6749

VL - 124

SP - 862

EP - 864

JO - Journal of Allergy and Clinical Immunology

JF - Journal of Allergy and Clinical Immunology

IS - 4

ER -

Thymic stromal lymphopoietin induction by polyinosinic:polycytidylic acid in human keratinocytes is preferentially mediated through protein kinase R and retinoid-inducible gene I and not Toll-like receptor 3

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